anti got1 Search Results


got1  (Atlas Antibodies)
90
Atlas Antibodies got1

Got1, supplied by Atlas Antibodies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+got1/Anti-GOT1/pmc08595646-549-12-34
Average 90 stars, based on 1 article reviews
got1 - by Bioz Stars, 2026-09
90/100 stars
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got1  (Boster Bio)
91
Boster Bio got1
<t>GOT1</t> inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.
Got1, supplied by Boster Bio, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+got1/Anti-GOT1+Antibody/pmc09641271-118-15-18
Average 91 stars, based on 1 article reviews
got1 - by Bioz Stars, 2026-09
91/100 stars
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antibody anti got1  (Synthego Inc)
86
Synthego Inc antibody anti got1
<t>GOT1</t> inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.
Antibody Anti Got1, supplied by Synthego Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+got1/anti+antibody+got1/10__7554_slash_elife__90024__3-166-81-95
Average 86 stars, based on 1 article reviews
antibody anti got1 - by Bioz Stars, 2026-09
86/100 stars
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anti-got1  (Abnova)
90
Abnova anti-got1
<t>GOT1</t> inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.
Anti Got1, supplied by Abnova, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+got1/anti+got1/pmc06941320-79-0-6
Average 90 stars, based on 1 article reviews
anti-got1 - by Bioz Stars, 2026-09
90/100 stars
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plasma glutamate oxaloacetase transaminase (got1)  (Merck KGaA)
90
Merck KGaA plasma glutamate oxaloacetase transaminase (got1)
<t>GOT1</t> inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.
Plasma Glutamate Oxaloacetase Transaminase (Got1), supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+got1/anti+got1+antibody++clone+1f5+2/pm37755804-102-1-22
Average 90 stars, based on 1 article reviews
plasma glutamate oxaloacetase transaminase (got1) - by Bioz Stars, 2026-09
90/100 stars
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goat anti-got1 (aa 22-35) antibody  (Angio-Proteomie)
N/A
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rabbit anti-human got1 polyclonal antibody  (Cusabio)
N/A
Rabbit anti-Human GOT1 Polyclonal Antibody
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anti-got1 monoclonal antibody, clone 5h5  (Creative BioMart)
N/A
Glutamic-oxaloacetic transaminase is a pyridoxal phosphate-dependent enzyme which exists in cytoplasmic and mitochondrial forms, GOT1 and GOT2, respectively. GOT plays a role in amino acid metabolism and the urea and tricarboxylic acid cycles. The two
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anti-got1 monoclonal antibody, clone fqs23256  (Creative BioMart)
N/A
Plays a key role in amino acid metabolism.Store at +4°C short term (1-2 weeks). Upon delivery aliquot. Store at -20°C long term. Avoid freeze / thaw cycle.http://www.creative-diagnostics.com/Anti-GOT1-MAb-165053-144.htm
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goat anti-got1 (aa 157-167) antibody  (Angio-Proteomie)
N/A
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anti-got1 antibody (biotin)  (St Johns Laboratory)
N/A
Goat polyclonal to GOT1 (Biotin). Conjugation note: Biotin Application note: ELISA, WB, IHC Reactivity note: Human
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anti-got1 (aa 157-167) polyclonal antibody  (Creative BioMart)
N/A
Plays a key role in amino acid metabolism.Shipped at 4°C. Upon delivery aliquot and store at -20°C. Avoid freeze / thaw cycles.http://www.creative-diagnostics.com/Anti-GOT1-PAb-207889-147.htm
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Image Search Results


Journal: Cell Reports

Article Title: The hepatic compensatory response to elevated systemic sulfide promotes diabetes

doi: 10.1016/j.celrep.2021.109958

Figure Lengend Snippet:

Article Snippet: Primary antibodies used were; TST, Rabbit, GeneTex, GTX114858, MPST, Rabbit, Abcam, Ab224043, GOT1, Rabbit, Abcam, ab170950, GSTT1, Rabbit, Proteintech, 15838-1-AP, MAT1A, Rabbit, Abcam, ab129176, BHMT Rabbit, Proteintech, 15965-1-AP, CSAD, Rabbit, Abcam, ab91016, PPCS Rabbit, Atlas Antibodies, HPA031361.

Techniques: Recombinant, Protease Inhibitor, Marker, Staining, Sequencing, Modification, Electron Microscopy, Cholesterol Assay, Multiplex sample analysis, Knock-Out, Gene Expression, Software

GOT1 inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.

Journal: Frontiers in Oncology

Article Title: Shikonin induces ferroptosis in multiple myeloma via GOT1-mediated ferritinophagy

doi: 10.3389/fonc.2022.1025067

Figure Lengend Snippet: GOT1 inhibition contributes to SHK-induced ferroptosis in MM. (A) Common targets of SHK and ferroptosis. (B) The binding energy of SHK bind with potential targets. (C) Molecular docking between SHK and GOT1. (D) The comparative study of GOT1 expression between healthy people and MM patients in GSE39754 database. (E) The comparative study GOT1 expression among healthy people and MGUS, SMM and MM patients in GSE6477 database. (F, G) The K-M survival curves analysis of GSE4452 and GSE4581 databases. (H) The expression of GOT1 influenced by SHK treatment for 24 h in RPMI 8226 and U266 cell lines. (I) The effects of DFOM on SHK regulated GOT1 expression. **p < 0.01, ***p < 0.001, ##p < 0.01.

Article Snippet: The samples were then hybridized with antibodies against glutathione peroxidase 4 (GPX4; 1:100, bm5231, Boster), GOT1 (1:100, bm5408, Boster), LC3B (1:2000, ab51520, Abcam), FTH1 (1:100, bm5356, Boster), NCOA4 (1:200, a5695, Abclonal), or 4HNE (1:25, ab51520, Abcam).

Techniques: Inhibition, Binding Assay, Expressing

GOT1 inhibition primes for ferroptosis by promoting ferritinophagy. (A, B) The effects of SHK on protein level of FTH1 and NCOA4 were determined in the presence or absence of DFOM. (C) The protein level of ATG5, ATG7, ATG16L1, SQSTM1 and LC3B was examined after treatment with SHK for 24 h. (D) The effects of SHK on protein level of SQSTM1, LC3B, SLC7A11, GPX4, NCOA4 and FTH1 were determined in the presence or absence of CQ. Data are presented as the mean ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, compared to control; ## p < 0.01, ### p < 0.001, compared to SHK.

Journal: Frontiers in Oncology

Article Title: Shikonin induces ferroptosis in multiple myeloma via GOT1-mediated ferritinophagy

doi: 10.3389/fonc.2022.1025067

Figure Lengend Snippet: GOT1 inhibition primes for ferroptosis by promoting ferritinophagy. (A, B) The effects of SHK on protein level of FTH1 and NCOA4 were determined in the presence or absence of DFOM. (C) The protein level of ATG5, ATG7, ATG16L1, SQSTM1 and LC3B was examined after treatment with SHK for 24 h. (D) The effects of SHK on protein level of SQSTM1, LC3B, SLC7A11, GPX4, NCOA4 and FTH1 were determined in the presence or absence of CQ. Data are presented as the mean ± SD. * p < 0.05, ** p < 0.01, *** p < 0.001, compared to control; ## p < 0.01, ### p < 0.001, compared to SHK.

Article Snippet: The samples were then hybridized with antibodies against glutathione peroxidase 4 (GPX4; 1:100, bm5231, Boster), GOT1 (1:100, bm5408, Boster), LC3B (1:2000, ab51520, Abcam), FTH1 (1:100, bm5356, Boster), NCOA4 (1:200, a5695, Abclonal), or 4HNE (1:25, ab51520, Abcam).

Techniques: Inhibition, Control

SHK results in ferroptosis induction and exerts antitumor efficacy in vivo . (A) Representative image of RPMI 8226 xenograft tumors after SHK treatment. (B) Tumor volume in each group. (C) Tumor weight in each group. (D) Tumor Fe 2+ in each group. (E) Tumor ATP level in each group. (F) Tumor MDA level in each group. (G) Tumor GSH level in each group. (H) Tumor GSSG level in each group. (I) IHC staining were used to assess GPX4, 4HNE, GOT1, NCOA4, FTH1 and LC3B levels in tumor tissues. (J) Serum ALT in each group. (K) Body weight in each group. (L) HE stain in liver, kidney and heart of each group. Data are presented as the mean ± SD. ns, not significant; ** p < 0.01, *** p < 0.001.

Journal: Frontiers in Oncology

Article Title: Shikonin induces ferroptosis in multiple myeloma via GOT1-mediated ferritinophagy

doi: 10.3389/fonc.2022.1025067

Figure Lengend Snippet: SHK results in ferroptosis induction and exerts antitumor efficacy in vivo . (A) Representative image of RPMI 8226 xenograft tumors after SHK treatment. (B) Tumor volume in each group. (C) Tumor weight in each group. (D) Tumor Fe 2+ in each group. (E) Tumor ATP level in each group. (F) Tumor MDA level in each group. (G) Tumor GSH level in each group. (H) Tumor GSSG level in each group. (I) IHC staining were used to assess GPX4, 4HNE, GOT1, NCOA4, FTH1 and LC3B levels in tumor tissues. (J) Serum ALT in each group. (K) Body weight in each group. (L) HE stain in liver, kidney and heart of each group. Data are presented as the mean ± SD. ns, not significant; ** p < 0.01, *** p < 0.001.

Article Snippet: The samples were then hybridized with antibodies against glutathione peroxidase 4 (GPX4; 1:100, bm5231, Boster), GOT1 (1:100, bm5408, Boster), LC3B (1:2000, ab51520, Abcam), FTH1 (1:100, bm5356, Boster), NCOA4 (1:200, a5695, Abclonal), or 4HNE (1:25, ab51520, Abcam).

Techniques: In Vivo, Immunohistochemistry, H&E Stain

SHK inhibited MM development by regulating GOT1 to activate ferritinophagy and further to promote ferroptosis in MM. (Created with BioRender.com ).

Journal: Frontiers in Oncology

Article Title: Shikonin induces ferroptosis in multiple myeloma via GOT1-mediated ferritinophagy

doi: 10.3389/fonc.2022.1025067

Figure Lengend Snippet: SHK inhibited MM development by regulating GOT1 to activate ferritinophagy and further to promote ferroptosis in MM. (Created with BioRender.com ).

Article Snippet: The samples were then hybridized with antibodies against glutathione peroxidase 4 (GPX4; 1:100, bm5231, Boster), GOT1 (1:100, bm5408, Boster), LC3B (1:2000, ab51520, Abcam), FTH1 (1:100, bm5356, Boster), NCOA4 (1:200, a5695, Abclonal), or 4HNE (1:25, ab51520, Abcam).

Techniques: